SNORD6 and cervical cancer: In conclusion, this study confirms that SNORD6 plays a promoting role in cervical carcinogenesis, which acts as a “hub” to promote the formation of the p53 ubiquitination degradation complex E6/ E6AP/ p53 and then accelerates p53 protein degradation, leading to the abnormal regulation of p53 in the cell cycle and apoptosis, then promotes the development of cervical cancer (Fig. 8).