In conclusion, this study confirms that SNORD6 plays a promoting role in cervical carcinogenesis, which acts as a “hub” to promote the formation of the p53 ubiquitination degradation complex E6/ E6AP/ p53 and then accelerates p53 protein degradation, leading to the abnormal regulation of p53 in the cell cycle and apoptosis, then promotes the development of cervical cancer (Fig. 8). This evidence concerns the gene SNORD6 and cervical carcinoma.