Evaluation of ICAM-1 and PECAM-1, potential biomarkers of neuronal and endothelial damage in BCS, demonstrated a significant increase in the levels of these molecules in patients with manifestations of vestibulo-atactic syndrome, which, in turn, suggests the leading role of endothelial dysfunction in the development of this pathology [25,53]. This evidence concerns the gene ICAM1 and endothelial dysfunction.