Inferring from these results, the TRIM molecules tested in this paper, are very likely to perform, as yet unknown functions, and in conjunction with Chen et al. studies, verifying the influence of TRIM47 on the oncogenesis of glioma cells via the Wnt/β-catenin pathway [55], the role of TRIM molecules with tumor research is promising. The gene discussed is TRAT1; the disease is central nervous system cancer.