Mechanistically, LncRNA STEAP3 antisense RNA 1 (STEAP3-AS1) interacted competitively with the YTHDF2, resulting in the disassociation of YTHDF2 with Six-transmembrane epithelial antigen of the prostate 3 (STEAP3) mRNA, which escaped the fate of degradation mediated by m6A modification and elevated the protein expression of STEAP3, thus finally activation of Wnt/β-catenin signaling to promote colorectal cancer progression [93]. The gene discussed is YTHDF2; the disease is colorectal cancer.