Mechanistically, the METTL14-mediated m6A modification on LncRNA HCG11 enhanced its nuclear exportation, thus recruiting IGF2BP2 to target Large Tumor Suppressor Kinase 1 (LATS1) mRNA to enhance the stability and promote the translation of LATS1, finally influencing the growth of lung adenocarcinoma [32]. This evidence concerns the gene METTL14 and lung adenocarcinoma.