The molecular and cellular pathological outcomes during MS progression are characteristic hallmarks of the myelin oligodendrocyte glycoprotein (MOG)35–55 induced animal models of experimental autoimmune encephalomyelitis (EAE) (Mendel et al., 1995; Hasselmann et al., 2017; Kipp et al., 2017). This evidence concerns the gene MOG and myeloid sarcoma.