To verify that TCN‐PM displays its antiproliferation effect against FLT3‐ITD AML cells through STAT5, we then performed a microscale thermophoresis (MST) assay to measure the direct binding affinity between TCN‐PM and STAT5B, which is one of the two STAT5 proteins known to play important roles in the pathogenesis of hematological malignancies.4 This evidence concerns the gene FLT3 and acute myeloid leukemia.