Proteins such as cell adhesion molecule 2 (CADM2), gap junction alpha-1 protein (Connexin-43), protein kinase C and casein kinase substrate in neurons protein 1 (Syndapin-1), Tenascin-N, Dysferlin-1 with well-known roles in cell adhesion and migration in Gliomas were found to be dysregulated in the comparison (Figure 2A). The gene discussed is TNN; the disease is glioma.