The results of GO analysis demonstrated that these GAs that vanished after NACT were particularly gathered in CD209, FLT3, down‐regulation of ERBB2 and ERBB3 signaling, regulation of TP53 activity through acetylation, acute myeloid leukemia, transcriptional regulation by AP‐2, and homology‐directed repair (HDR) through single‐strand annealing (SSA). The gene discussed is ERBB3; the disease is acute myeloid leukemia.