IL10 and infection: In contrast to the results described during LCDV-Sa natural or experimental infections, the vaccine induced an inflammatory process by the overexpression of pro-inflammatory genes (il1β, il6, casp1, ck3, and ck10), and the down-regulation of the anti-inflammatory interleukin 10 (il10), also driving the production of specific neutralizing antibodies, conferring a possible protective state against LCDV-Sa.