TGFB1 and acute myeloid leukemia: Gene expression profiles indicated that compared with biCEBPA/‐like IKZF1, the HOXA/B, HSPC expression signature, proto‐oncogene pathways including VEGF, B‐cell receptor, NOTCH, MAPK, WNT, TGF‐beta, Rap1 signalling pathways were significantly upregulated in the IKZF1 N159S‐positive AML.