NFKB1 and Epstein-Barr virus infection: Notably, 4134/Late cells were found to be more resistant to NF-κB p65 inhibition compared to BL41 cells, an effect that is likely due to EBV infection, as the viral latent membrane protein 1 (LMP1) regulates NF-κB p65 activation [86] and nuclear translocation [87].