Considering the inverse relationship between the expression of VDAC1P4, VDAC1P11 and in particular VDAC1P8 and VDAC1, these pseudogenes in AML could function as competing-endogenous RNA (ceRNA), or rather as a lncRNA able to generate siRNA acting on the transcript of the parental gene, as described in [18]. This evidence concerns the gene VDAC1P11 and acute myeloid leukemia.