In addition, DHA can also independently inhibit the proliferation of cervical cancer cells by ferroptosis, which is reflected in DHA-based induction of ferroptosis; this mechanism involves the accumulation of lipid ROS and the consumption of GPX4, which further exacerbates ferroptosis by promoting nuclear receptor coactivator 4 (NCOA4)-mediated ferritinophagy [100]. Here, NCOA4 is linked to cervical cancer.