FBLN7‐knockout (Fbln7−/−) mice do not show obvious developmental or health defects seen in the absence of other fibulin genes in animal models, such as bleeding,[27] retinopathy,[28] macular dystrophy,[29] and defective elastic fiber formation.[30] Wild‐type (Fbln7+/+) and Fbln7−/− mice were randomly subjected to MI surgery or sham operation. Here, FBLN7 is linked to retinal disorder.