It was shown that Fer‐1 prevents glutamate induced ferroptosis cell death in postnatal rat brain, suggesting that the inhibition of ferroptosis could be exploited to preserve neuronal cells and protect organisms from specific oxidative, iron‐dependent neurodegeneration, such as AD, PD, and strokes.[1] The existing evidence suggests a strong correlation between ferroptosis and neurodegenerative diseases and stroke through a shared mechanism involving dysregulation of iron metabolism, induction of LPO, and inhibition of GSH/GPX4 axis. This evidence concerns the gene GPX4 and Parkinson disease.