AKT1 and neoplasm: When comparing the untreated MAS98.12 and HbCx39 models, we found that Akt‐pS473, Akt‐pT308, Akt1‐pS473, and Akt2‐pS474 all had an increased expression in MAS98.12 (Fig. 3, Fig. S2), which may explain the effect of LXR activation on tumor growth observed in this model.