CFTR and Duchenne muscular dystrophy: Aminoglycoside antibiotics have been shown to induce translational readthrough in various genes with nonsense mutations, including TP53, PTEN, APC (adenomatous polyposis coli), ATM (ataxia telangiectasia mutated), CFTR (cystic fibrosis transmembrane conductance regulator), and DMD (Duchenne muscular dystrophy) [19–23].