β2AR deficiency leads to increased numbers of IL-5+ and IL-13+ ILC2s and protects the host from gastrointestinal helminth Nippostrongylus brasiliensis (N. brasiliensis) infection, while β2AR treatment inhibits ILC2-mediated type 2 immune responses and compromises the hosts protection against helminths [51]. Here, ADRB2 is linked to infection.