We found no driver mutations in TET2, DNMT3A, PPM1D, IDH1, IDH2, TP53, CHEK2, ASXL1, MBD4 or other genes associated with myelodysplasia or AML, and the patient has reported no haematological problems in over two decades of follow-up. This evidence concerns the gene MBD4 and Myelodysplasia.