Transcriptional profile analysis of CX3CR1highPD-L1low MG isolated from IFN-γ- and PBS-treated EAE mice revealed that genes with a pro-inflammatory role in MC, MG and EAE such as Cd38 (68–71, 109), Cfb (72, 108), Saa3 (75, 76), Inhba, Anpep (110, 111), and Apoc2 (112) were down-regulated by IFN-γ. Here, APOC2 is linked to myasthenia gravis.