Previous study has found that treatment with PDGF‐BB restores neural function by regulating astrocytic PDGFRβ signaling in STAT3‐dependent manner after subarachnoid hemorrhage.[52] Moreover, the proliferation and differentiation of neural progenitor cells are differentially regulated by the JAK pathway.[53] Our in vitro experiments showed that exposure of NSCs to PDGF‐BB resulted in increased JAK2‐STAT3 signaling. Here, JAK2 is linked to subarachnoid hemorrhage.