Given the close relationship between baicalin, miR-124, TLR4, inflammation, and renal fibrosis, Zhang et al. (78) looked into the link between baicalin and the miR-124/TLR4/NF-κB axis and discovered that by inhibiting TLR4/NF-κB pathway and upregulating miR-124, baicalin alleviated renal fibrosis. The gene discussed is NFKB1; the disease is renal fibrosis.