found that M1‐like macrophage‐derived MiVs own superior advantageous features for tumor‐targeting delivery of chemotherapeutic drugs compared with Exos.[33] One reason is that MiVs are directly budded from the cell membrane, largely inherit the parental cell membrane properties, and exhibit CCL2/CCR2‐mediated tumor‐homing function. This evidence concerns the gene CCR2 and neoplasm.