HDAC9 and lung adenocarcinoma: Importantly, in contrast to HDAC-inhibiting (R)-lipoic acid and Vorinostat, (S)-lipoic acid did not lead to a substantial increase of α-Tubulin AcK40 or Histone H4 AcK5/8/12/16 in HEK293T cells or A549 lung adenocarcinoma cells at concentrations of up to 500 μM (Fig. 3b, Supplementary Fig. 3c).