Furthermore, to better understand potential population differences between these studies, we compared 3 DM1-associated splicing events (ATP2A1 exon 22, CLASP1 exon 20, and CLCN1 exon 7a) for control participants, pretraining participants, and posttraining participants with DM1 with published data on control and pretraining and posttraining individuals with DM1 (17). This evidence concerns the gene CLASP1 and myotonic dystrophy type 1.