Post-mortem histology showed that the number of F4/80+ total TAMs and MRC1+ tumor-supportive TAMs in the TME were decreased by Ccl21a knockdown in GBM, while activated MHC-II+ antigen-presenting cells (APCs) were unchanged (Fig. 6a-b, Supplementary Fig. 6c, d). Here, MRC1 is linked to glioblastoma.