In Parkinson's disease (PD) model, circSV2b overexpression can reduce oxidative stress injury, protect dopaminergic neuron loss, maintain nigrostriatal function, and improve motor defects through miR‐5107‐5p‐FOXK1 (fork‐head box protein K1)‐AKT1 (serine/threonine protein kinase) signaling pathways.126. Here, AKT1 is linked to Parkinson disease.