Interestingly, the antisense of TRAF3IP2, which is an lncRNA termed TRAF3IP2-AS1, is also vital in regulating psoriasis, especially in inhibiting the IL−17A-induced activation of NF-κB signalling and MAPK signalling by binding to SRSF10 and thereby blocking the subsequent recruitment of ACT1 [55]. The gene discussed is IL17A; the disease is psoriasis.