MARK1 and nasopharyngeal carcinoma: Ye et al. found that the microRNAs (miRNAs) miR‐24‐3p, miR‐891a, miR‐106a‐5p, miR‐20a‐5p, and miR‐1908 are over‐expressed in EVs extracted from patient sera or NPC cells, and by down‐regulating microtubule affinity regulating kinase 1 (MARK1) signaling these miRNAs can affect cell proliferation and differentiation.26