Analysis of microarray data indicates that the tyrosine phosphatase, SHP2, which has been demonstrated to increase migration of TNBC cells [56] and to have a role in promoting TNBC and basal-like breast cancer [57], and whose inhibition results in a basal-to-luminal transition [58], regulates expression of ACSL4 protein in MA-10 Leydig cells [59]. The gene discussed is ACSL4; the disease is breast carcinoma.