MO ethanolic extracts and its phytochemical derivative, α-pinene, may reduce cerebral infarct volume and MDA levels, while conferring further cereborpotective effects by increasing the levels of antioxidant enzymes in SOD, GSH-Px and CAT, which have the potential to reduce oxidative stress generated by ischemic stroke. The gene discussed is SOD1; the disease is ischemic stroke.