FCGR3A and sarcoidosis: In conclusion, the increased percentage of monocytes in the peripheral blood of SA versus TB patients, with their comparably high phagocytic activity, but different patterns of Fcγ and complement receptors with genetically coded different functional variants of CD16A (FcγRIIIa), could have resulted in different pathomechanisms and outcomes of SA and TB, even in the light of the suspected contribution of mycobacterial antigens in the development of sarcoidosis.