FCGR2A and tuberculosis: The persistent antigenemia and immunocomplexemia, with a decreased ability to process antigens and present them at the surface of monocytes, may contribute to the occurrence of the prolonged inflammatory response in SA patients, whereas the more effective IC-bound antigen engulfment (due to the increased presence of both FcγR and CR), processing, and presentation to T lymphocytes can translate into an exaggerated granulomatous immune response present in our patients with active tuberculosis.