In addition, because our NV848, NV914, and NV930 compounds have been shown to rescue both protein levels and functionality of the CFTR (Cystic Fibrosis Transmembrane Conductance Regulator) chloride channel [14], mutated in CF, we also tested for CFTR protein cellular localization in IB3.1 cystic fibrosis cells [18] upon FTSJ1 transfection. This evidence concerns the gene CFTR and cystic fibrosis.