We noted a significant upregulation between MYD88 and CCL20 (G60); IFNβ (G90); NF-kB, IL18 (G120); NF-kB, CCR2, IL1b, IL18 (GF120); and CCR2 (GR120) in the thoracic aorta during the inflammatory process of atherosclerosis (Table 3). The gene discussed is IL1B; the disease is atherosclerosis.