Wang and Chang [3] demonstrated that maspin suppresses cell invasion and migration in gastric cancer through inhibiting EMT (epithelial–mesenchymal transition) and angiogenesis via the ITGB1/FAK pathway; results were based on the immunohistochemical determination of maspin correlated with the expression of ITGB1, FAK, E-cadherin, vimentin, D2-40, and CD34. Here, SERPINB5 is linked to gastric cancer.