ATP6V1A and acute myeloid leukemia: A study performed on a DNMT3A R878H conditional knock-in mouse model, used to predict the specific long non-coding RNAs (lncRNAs) regulated by DNMT3A mutations in AML, first identified 23 differentially expressed lncRNAs, then the downstream target genes regulated by these lncRNAs, including ATP6V1A, a critical autophagy-related gene, the overexpression of which is associated with poor prognosis in AML [167].