The mainly observed upregulation of Cables1 and DCHS1 during dormancy entry and exit in the primary cultures, and the correlations found with CCRL1, SLFN13, and SKI seem contradictory considering the tumor-restricting functions of Cables1 and DCHS1, both located on the same chromosome 18q [22,45], as postulated in the literature. The gene discussed is DCHS1; the disease is neoplasm.