To evaluate the relevance of CCRL1, SLFN13, SKI, Cables1, and DCHS1 in GBM progression, we first examined their gene expression under TMZ-promoted dormancy entry and exit in different GBM cell lines (LN229 and U251) and primary cultures (PCa and PCb), respectively, using our previously established in vitro model with sole-TMZ stimulation. The gene discussed is CABLES1; the disease is posterior cortical atrophy.