TASL and neoplasm: We further comprehensively analyzed the correlation of TASL expression with the level of infiltration of various TIICs in the TME of LGG, LUAD, and SKCM, and the results implied that TASL may mediate the immune escape of the “cold” tumor LGG by inducing the conversion of Th1 cells to Th2 cells and the conversion of M1 macrophages to M2 macrophages.