The elevated lipid peroxidation level caused by BAP31 knockdown was dramatically attenuated by VBIT-12 (Supplementary Fig. S5H and Fig. 5J, K), which suggested that VDAC1 oligomerization inhibitor prevented GC cells from BAP31 knockdown-induced VDAC1 oligomerization and ferroptosis. The gene discussed is VDAC1; the disease is gastric cancer.