Finally, although KIF13A did not recruit MBNL in our kinesin tail screen, exon 32 is believed to be an MBNL splicing target and is a robust biomarker of disease status in DM1—functions of exon 32 are unknown, and a potentially fruitful line of investigation may be to probe its roles in the pathways discussed here102,103. The gene discussed is MBNL1; the disease is myotonic dystrophy type 1.