For example, factors including age, increased parity, higher pre-conception body mass index (BMI), family history of diabetes, insulin therapy during pregnancy, degree of hyperglycemia during pregnancy (higher area under the curve (AUC) of glucose, higher fasting plasma glucose (FPG)), and impaired pancreatic β-cell function were consistently found to be associated with postpartum glucose intolerance [11, 13, 19–23]. The gene discussed is INS; the disease is diabetes mellitus.