To test whether the LSTase activity of CPT1A that stabilizes MFF may contribute to cell proliferation in ovarian cancer cells, we exogenously overexpressed H473A in the background of CPT1A knockdown and found that CPT1A WT rescued mitochondrial fission and cellular ATP production, but H473A did not (Fig. 5i, Supplementary Fig. 5c, d). The gene discussed is CPT1A; the disease is ovarian carcinoma.