In acute myeloid leukemia, IGF2BP2 enhances the mRNA stability and translation initiation of m6A-containing targets GPT2, SLC1A5, and MYC by employing PABPC1 and eukaryotic translation initiation factor (eIF) complexes eIF4A, respectively, facilitating glutamine uptake and metabolism for tumor cell stemness and development [83]. Here, IGF2BP2 is linked to neoplasm.