GATA6 and gastric cancer: CCK-8 assays revealed that the increase in cell viability caused by GATA6-AS1 deficiency could be fully reversed by cotransfection with miR-543 inhibitors (Figure 4(b)), indicating that miR-543 reversed the increase in cell viability induced by GATA6-AS1 knockdown and miR-543 silencing represses cell viability in GC cells.