The scRNA-seq data analyzed in this study was obtained from surgically isolated murine glomeruli from murine in the development of GN induced by injury, not from the whole kidney, and GN is different from lupus; however, the downstreaming cell-cell communication analysis between neutrophils and B cells and the coculture experiment showed consistent result that neutrophil-derived IL-6 could promote ferroptosis resistance in lupus kidney B cells, suggesting this could possibly be a general phenomenon in the development of glomerular injury. This evidence concerns the gene IL6 and ganglioneuroma.