Additionally, NPC‐infiltrating DN B cells interacted with exhausted T cells within the NPC TME via the CXCL13‐CXCR5 axis, suggesting that at least a portion of DN B cells expressed CXCR5 and were recruited into NPC tumors via CXCL13‐mediated chemotaxis. The gene discussed is CXCR5; the disease is nasopharyngeal carcinoma.