that focused on hepatocellular carcinoma reported that YAP/TAZ, as a transcriptional coactivator, formed a complex with TEADs that indirectly bound the TEAD sequence in the SLC7A11 gene promoter, ultimately leading to upregulating the content of SLC7A11 and inhibiting the development of ferroptosis (138). Here, SLC7A11 is linked to hepatocellular carcinoma.