IL-17 has been shown to drive a Th1-mediated response to infection in similar fashion, with C. muridarum-infected il17ra-/- and il17-/- mice exhibiting diminished recruitment of neutrophils and/or macrophages (Scurlock et al., 2011; Andrew et al., 2013), likely due to the attenuated activity of known IL-17-producing cell types at the infection site (e.g. Th17, γδ+ T, NK cells) (Gaffen et al., 2014; Monin and Gaffen, 2018). Here, IL17A is linked to infection.