In mouse models of congenital infection, Type II IFNγ‐mediated maternal immunity simultaneously promotes parasite clearance and prevents vertical transmission; however, the production of excessive IFNγ from T. gondii infection can also have dire consequences for the developing fetus, such as stillbirth and spontaneous abortion (Shiono et al, 2007; Pappas et al, 2009; Senegas et al, 2009). Here, IFNG is linked to Stillbirth.