Abnormal SLC37A2 causing bone dysplasia or osteonecrosis has been reported37, 38 In our study the expression of the gene/protein at E85 was significantly higher than that at E45, and the protein expression was more than 40 times higher, suggesting that G6P and Pi exchange was active and metabolism was enhanced in E85 condylar cells. Here, SLC37A2 is linked to osteonecrosis.